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I've been on Coumadin since I was diagnosed with pulmonary embolism and it seems to work. I get my INR tested regularly. It's currently 3.0 and it's the best result I've had so far.
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I'm taking this medicine for my blood clots. It seems to work and keeps me alive. So far so good.
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It also does not reverse ischemic tissue damage (tissue damage caused by a lack of blood flow to the area).
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Taking this capsule needs a lot of self relief management.
It is better to discuss whatever questions you have with your doctor and follow his recommendations.
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Best to double check with the doctor's office, if you have an infection brewing, you need to see a doctor anyway.
INR and risk of hemorrhage) due to vitamin K deficiency, scurvy, malnutrition or cachexia, small body size, hepatic dysfunction, moderate to severe renal impairment, fever, hyperthyroidism, infectious disease, carcinoma, collagen disease, CHF, diarrhea, biliary obstruction, old age, debility, menstruation and menstrual disorders, radiation therapy, initial hypoprothrombinemia, and decreased clearance of warfarin as a result of variations in genes responsible for warfarin metabolism.
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It is recommended that for healthy individuals, all the nutrients you need should come from the foods you eat.
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LIKE SO MANY OTHERS ARE FORCED TO TAKE COUMADIN(WARFARIN) FOR LIFE.
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Aspirin and bisoprolol isn't as effective because aspirin doesn't work on the type of clots that form in a-fib.
Newer oral anticoagulants should be used as first-line agents to prevent thromboembolism in patients with atrial fibrillation and risk factors for stroke or thromboembolism.
Anyway, here is my present question: is there something I could have done and could now do that would have prevented the stent from the fibrosis?
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The discharge date is the day that the patient is discharged from your institution's acute care unit OR the date of the patient's expiration OR the date of the patient's discharge OR date patient left against medical advice (AMA) OR date of transfer to, a rehabilitating, skilled nursing, or hospice unit in your institution OR transfer to an acute in-patient unit outside of your own institution, even if that hospital is affiliated with your own OR expired.
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No one has ever mentioned to me any thought of taking warfarin for this purpose.
And if so, should I talk to my doctor about having the genetic testing?
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Every time I work up in the first 8 hours, my headache was still there.
Taking vitamins B-6 or B-12, TMG or folic acid could help to regulate your levels of homocysteine, an amino acid that may relate to risks of atherosclerosis or heart disease.
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This should also be considered for patients with acute HIT without thrombosis (isolated HIT), because there is a high risk for subsequent clinically evident thrombosis in these patients.
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The doctors say sometimes this may happen for no reason.
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Doctors have perscribed Migraine meds and muscle relaxers, sometimes it does help but was recently started on Neurontin works a little, but when taking higher does of Coumadin nothing helps, but sometimes a visit to the ER for pain management.
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Clinicians need to balance the individual's risk of thromboembolism, based on the medical history and surgical procedure, and risk of bleeding when determining what is optimal in persons in this moderate-risk category.
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Note that the S-isomer is more potent than the R-isomer; thus, drugs that inhibit S-isomer metabolism have a greater effect on PT.
Happy to find your site as I try to first approach things naturally.
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Some foods, vitamins and herbal supplements are high in Vitamin K and should be avoided in large quantities while on this medication.
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Are there any natural alternatives that would work just as well?
In most patients, warfarin should be initiated as a maintenance dosage of 5 mg daily.
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